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    <!-- http://purl.obolibrary.org/obo/HP_0032054 -->

    <Class rdf:about="http://purl.obolibrary.org/obo/HP_0032054">
        <rdfs:label>Focal cortical dysplasia type III</rdfs:label>
    </Class>
    


    <!-- http://purl.obolibrary.org/obo/HP_0032056 -->

    <Class rdf:about="http://purl.obolibrary.org/obo/HP_0032056">
        <rdfs:label>Focal cortical dysplasia type IIIb</rdfs:label>
        <rdfs:subClassOf rdf:resource="http://purl.obolibrary.org/obo/HP_0032054"/>
        <dcterms:date rdf:datatype="http://www.w3.org/2001/XMLSchema#dateTime">2018-09-16T11:18:31Z</dcterms:date>
        <ns2:IAO_0000115>A subtype of focal cortical dysplasia type III that is characterized by altered architectural (cortical dyslamination, hypoplasia without six-layered structure) and/or cytoarchitectural composition (hypertrophic neurons) of the neocortex, which occur adjacent to glial or glioneuronal tumor.</ns2:IAO_0000115>
        <rdfs:comment>This type of focal cortical dysplasia is associated with glial or glioneuronal tumors (Ganglioglioma, Dysembryoplastic Neuroepithelial Tumor (DNT, syn. DNET) or other epilepsy-associated neoplasms. It is important to exclude tumor infiltration in areas of cortical abnormalities before establishing the diagnosis of FCD. The etiology and pathogenesis of FCD Type IIIb remains to be determined, but is likely an acquired process.</rdfs:comment>
        <dcterms:creator rdf:resource="https://orcid.org/0000-0002-0736-9199"/>
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