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        <rdf:type rdf:resource="http://www.w3.org/2002/07/owl#TransitiveProperty"/>
        <rdfs:label>has part</rdfs:label>
        <rdfs:label xml:lang="en">has part</rdfs:label>
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    <!-- http://purl.obolibrary.org/obo/RO_0000052 -->

    <ObjectProperty rdf:about="http://purl.obolibrary.org/obo/RO_0000052">
        <rdf:type rdf:resource="http://www.w3.org/2002/07/owl#FunctionalProperty"/>
        <rdfs:label xml:lang="en">characteristic of</rdfs:label>
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        <rdfs:label>has modifier</rdfs:label>
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    <!-- http://purl.obolibrary.org/obo/GO_0042065 -->

    <Class rdf:about="http://purl.obolibrary.org/obo/GO_0042065">
        <rdfs:label>glial cell growth</rdfs:label>
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    <!-- http://purl.obolibrary.org/obo/HP_0001507 -->

    <Class rdf:about="http://purl.obolibrary.org/obo/HP_0001507">
        <rdfs:label>Growth abnormality</rdfs:label>
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    <!-- http://purl.obolibrary.org/obo/HP_0002171 -->

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        <rdfs:label>Gliosis</rdfs:label>
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        <rdfs:comment>Gliosis generally occurs as a response to tissue damage. Gliosis appears bright on T2 scans upon magnetic resonance imaging, unlike encephalomalacia which follows CSF signal on all sequences. Glial cells, the non-neuronal component of the central nervous system, are divided into microglia and macroglia. The latter are in turn divided into astrocytes, oligodendrocytes, and ependymal cells. The astrocytes and the microglia are the glial cells predominantly responsible for tissue response to injury.</rdfs:comment>
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    <!-- http://purl.obolibrary.org/obo/HP_0100705 -->

    <Class rdf:about="http://purl.obolibrary.org/obo/HP_0100705">
        <rdfs:label>Abnormal glial cell morphology</rdfs:label>
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    <!-- http://purl.obolibrary.org/obo/PATO_0000460 -->

    <Class rdf:about="http://purl.obolibrary.org/obo/PATO_0000460">
        <rdfs:label>abnormal</rdfs:label>
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    <Class rdf:about="http://purl.obolibrary.org/obo/PATO_0000912">
        <rdfs:label>increased rate</rdfs:label>
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