has part lipidosis [toxic course] hyperfunction of fatty acid biosynthesis [Lipidosis] hypofunction of lipid degradation [Lipidosis] lipid metabolism imbalance [Lipidosis] hyperfunction of lipid biosynthesis [Lipidosis] hyperfunction of lipid synthetic gene expression [Lipidosis] hypofunction of mitochondrial fatty acid beta-oxidation [Lipidosis] USF1 activation [lipidosis] SREBP activation [lipidosis] malfunctioning of insulin signaling [lipidosis] LXRa activation [lipidosis] ChREBP activation [lipidosis] lipid strorage in hepatocyte [Lipidosis] increasing uptake amount of mitochondrial calcium [Lipidosis] negative regulation of mitochondrial beta oxidation [lipidosis] Farnesoid X Receptor inactivation [lipidosis] hyperfunction of glucose transport to liver [Lipidosis] hypofunction of triglyceride transport from hepatocyte to extrahepatic [Lipidosis] insulin resistance (process) [Lipidosis] non-alcoholic fatty liver disease PMID:24133660 PMID:26962021 PMID:30642126 The totality of all processes through which non-alcoholic fatty liver disease (NAFLD) is realized. hypofunction of lipoprotein biosynthesis [Lipidosis] overeating [NAFLD] increasing fatty acid synthesis substrate [lipidosis] converting to acetyl CoA [NAFLD] increasing amount of carbohydrate [NAFLD] increase in lipid inflow into hepatocyte [NAFLD] increaing in fatty acid inflow into hepatocyte [NAFLD]