Toxic Process Ontology
645 terms(s) returned
| Term Type: | Record: 1 to 50 of 645 Records | Page: 1 of 13, First Previous Next Last | Show Records Per Page |
- I-kappaB -NfkB inhibitor [ER stress - inflammation]
- I-kappaB phosphorylation
- I-kappaB phosphorylation [ER stress - inflammation - IRE1-NfkB pathway]
- I-kappaB phosphorylation [NfKB pathway]
- IFN alpha inactivation
- IFN alpha inactivation [hepatocarcinogenesis]
- IGFBP1 (human)[ER stress]
- IGFBP1 (mol)
- IGFBP1 activation
- IGFBP1 activation [ER stress]
- IGFBP1 gene expression by ATF4 [ER stress]
- IGFBP3 (human) [hepatocarcinogenesis]
- IGFBP3 (mol)
- IGFBP3 - inactive form (human) [hepatocarcinogenesis]
- IGFBP3 inactivation
- IGFBP3 inactivation [hepatocarcinogenesis]
- IHH (canonical) [hepatocarcinogenesis]
- IKB
- IKK activation
- IKK activation [ER stress - inflammation - IRE1-TRAF2-NfkB pathway]
- IKK activation [NfKB pathway]
- IL-1 (canonical)[Fibrosis]
- IL-10 (canonical)[Glutathione depletion]
- IL-1B (canonical)[Glutathione depletion]
- IL-1B (canonical)[lipidosis]
- IL-1B (canonical)[pyroptosis]
- IL-1B(canonical) [hepatocarcinogenesis]
- IL-1B(canonical) [mitochondrial disorder]
- IL-1b (mol)
- IL-1b activator role
- IL-1beta processing
- IL-1beta processing [lipidosis]
- IL-1beta production [lipidosis]
- IL-1beta production [mitochondrial disorder]
- IL-2 gene expression
- IL-2 production inducer
- IL-2 signaling (primitive)
- IL-2 signaling (primitive) [Phospholipidosis (excessive defense) - apoptosis inhibition ]
- IL-2 signaling (primitive) [Phospholipidosis (excessive defense) - cell cycle arrest ]
- IL-2 signaling (primitive) [Phospholipidosis (excessive defense) - inflammation ]
- IL-2 signaling (primitive) [Phospholipidosis (excessive defense) ]
- IL-4 (canonical)[Glutathione depletion]
- IL-6 (canonical)[ER stress]
- IL-6 (canonical)[Fibrosis]
- IL-6 (mouse)[ER stress]
- IL-6 - inactive form (human)[hepatocarcinogenesis]
- IL-6 - inflammatory cytokine (canonical)[ER stress - inflammation -ATF4 pathway]
- IL-6 - inflammatory cytokine (canonical)[ER stress - inflammation -XBP1 pathway]
- IL-6 - inflammatory cytokine (canonical)[Glutathione depletion]
- IL-6 inactivation
